换一批
换一批Macrophage-inflammatory protein-3alpha mediates epidermal growth factor receptor transactivation and ERK1/2 MAPK signaling in Caco-2 colonic epithelial cells via metalloproteinase-dependent release of amphiregulin.
第一作者：
Sarah,Keates
第一单位：
Division of Gastroenterology, Beth Israel Deaconess Medical Center, Harvard Medical School, 330 Brookline Avenue, Boston, MA 02215, USA.
作者：
主题词
抗体, 单克隆(Antibodies, Monoclonal);Caco-2细胞(Caco-2 Cells);细胞增殖(Cell Proliferation);趋化因子CCL20(Chemokine CCL20);趋化因子, CC(Chemokines, CC);结肠(Colon);糖蛋白类(Glycoproteins);人类(Humans);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);肠黏膜(Intestinal Mucosa);巨噬细胞炎性蛋白质类(Macrophage Inflammatory Proteins);金属蛋白酶类(Metalloproteases);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);磷酰化(Phosphorylation);受体, CCR6(Receptors, CCR6);受体, 趋化因子(Receptors, Chemokine);信号传导(Signal Transduction);转录激活(Transcriptional Activation)
DOI
10.4049/jimmunol.178.12.8013
PMID
17548638
发布时间
2020-12-26
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