Deletion of N-glycosylation sites of hepatitis C virus envelope protein E1 enhances specific cellular and humoral immune responses.
第一作者:
Min,Liu
第一单位:
Department of Immunology, State Key Laboratory of Virology and Hubei Province Key Laboratory of Allergy and Immunology, Wuhan University School of Medicine, Wuhan 430071, China.
作者:
医学主题词
氨基酸序列(Amino Acid Sequence);动物(Animals);抗体生成(Antibody Formation);结合部位(Binding Sites);印迹法, 蛋白质(Blotting, Western);CD8阳性T淋巴细胞(CD8-Positive T-Lymphocytes);细胞系, 肿瘤(Cell Line, Tumor);细胞因子类(Cytokines);女(雌)性(Female);糖基化(Glycosylation);肝炎病毒属(Hepacivirus);免疫, 细胞(Immunity, Cellular);干扰素γ(Interferon-gamma);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);诱变, 定点(Mutagenesis, Site-Directed);突变(Mutation);重组蛋白质类(Recombinant Proteins);序列缺失(Sequence Deletion);疫苗, DNA(Vaccines, DNA);病毒包膜蛋白质类(Viral Envelope Proteins)
DOI
10.1016/j.vaccine.2007.07.003
PMID
17675185
发布时间
2008-11-21
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Vaccine
6572-80页
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