换一批
换一批TWEAK/Fn14 interactions are instrumental in the pathogenesis of nephritis in the chronic graft-versus-host model of systemic lupus erythematosus.
作者：
主题词
动物(Animals);抗体(Antibodies);自身抗体(Autoantibodies);炎症趋化因子类(Chemokines);慢性病(Chronic Disease);细胞因子类(Cytokines);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);移植物抗宿主病(Graft vs Host Disease);免疫球蛋白G(Immunoglobulin G);红斑狼疮, 系统性(Lupus Erythematosus, Systemic);狼疮肾炎(Lupus Nephritis);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);蛋白尿(Proteinuria);受体, 肿瘤坏死因子(Receptors, Tumor Necrosis Factor);敏感性与特异性(Sensitivity and Specificity);肿瘤坏死因子类(Tumor Necrosis Factors)
DOI
10.4049/jimmunol.179.11.7949
PMID
18025243
发布时间
2022-04-08
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