Implications of the progressive self-association of wild-type human factor H for complement regulation and disease.
作者:
主题词
氨基酸序列(Amino Acid Sequence);氨基酸取代(Amino Acid Substitution);结合部位(Binding Sites);补体因子H(Complement Factor H);二聚作用(Dimerization);基因表达调控(Gene Expression Regulation);溶血尿毒症综合征(Hemolytic-Uremic Syndrome);组氨酸(Histidine);人类(Humans);模型, 分子(Models, Molecular);分子序列数据(Molecular Sequence Data);分子量(Molecular Weight);视盘小疣(Optic Disk Drusen);多态现象, 遗传(Polymorphism, Genetic);蛋白质结合(Protein Binding);蛋白质结构, 三级(Protein Structure, Tertiary);散射, 辐射(Scattering, Radiation);同步加速器(Synchrotrons);超速离心法(Ultracentrifugation);X线(X-Rays)
DOI
10.1016/j.jmb.2007.11.015
PMID
18054958
发布时间
2021-10-27
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Journal of molecular biology
891-900页
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