换一批SIK2 can be activated by deprivation of nutrition and it inhibits expression of lipogenic genes in adipocytes.
第一作者：
Jing,Du
第一单位：
Brown Medical School, Department of Medicine, Hallett Center for Diabetes and Endocrinology, Providence, RI, USA.
作者：
医学主题词
3T3-L1细胞(3T3-L1 Cells);AMP活化蛋白激酶类(AMP-Activated Protein Kinases);乙酰CoA羧化酶(Acetyl-CoA Carboxylase);腺苷三磷酸(Adenosine Triphosphate);脂细胞(Adipocytes);氨基咪唑甲酰胺(Aminoimidazole Carboxamide);动物(Animals);剂量效应关系, 药物(Dose-Response Relationship, Drug);减量调节(Down-Regulation);电穿孔(Electroporation);酶激活(Enzyme Activation);酶激动剂类(Enzyme Activators);基因表达调控, 酶学(Gene Expression Regulation, Enzymologic);基因, 报告(Genes, Reporter);葡萄糖(Glucose);脂肪生成(Lipogenesis);小鼠(Mice);多酶复合物(Multienzyme Complexes);磷酰化(Phosphorylation);启动区, 遗传(Promoter Regions, Genetic);RNA干扰(RNA Interference);RNA, 小分子干扰(RNA, Small Interfering);硬脂酰CoA去饱和酶(Stearoyl-CoA Desaturase);胆固醇调节元件结合蛋白质1(Sterol Regulatory Element Binding Protein 1);时间因素(Time Factors);转染(Transfection);甘油三酯类(Triglycerides)
DOI
10.1038/oby.2007.98
PMID
18239551
发布时间
2021-12-03
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