Conditional FKBP12.6 overexpression in mouse cardiac myocytes prevents triggered ventricular tachycardia through specific alterations in excitation-contraction coupling.
作者:
主题词
动作电位(Action Potentials);肾上腺素能β激动剂(Adrenergic beta-Agonists);动物(Animals);钙信号(Calcium Signaling);心脏起搏, 人工(Cardiac Pacing, Artificial);儿茶酚胺类(Catecholamines);多西环素(Doxycycline);异丙肾上腺素(Isoproterenol);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);心肌收缩(Myocardial Contraction);肌细胞, 心脏(Myocytes, Cardiac);磷酰化(Phosphorylation);蛋白质构象(Protein Conformation);蛋白质加工, 转译后(Protein Processing, Post-Translational);重组融合蛋白质类(Recombinant Fusion Proteins);兰尼碱受体钙释放通道(Ryanodine Receptor Calcium Release Channel);肌浆网(Sarcoplasmic Reticulum);心动过速, 室性(Tachycardia, Ventricular);他克莫司结合蛋白质类(Tacrolimus Binding Proteins);增量调节(Up-Regulation)
DOI
10.1161/CIRCULATIONAHA.107.731893
PMID
18378612
发布时间
2013-11-21
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Circulation
1778-86页
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