Mechanisms of genesis of variant translocation in chronic myeloid leukemia are not correlated with ABL1 or BCR deletion status or response to imatinib therapy.
第一作者:
Steven,Richebourg
第一单位:
Laboratory of Medical Genetics, Hospital Jeanne de Flandre, University Hospital Regional Center-CHRU, Avenue Eugène Avinée, 59037 Lille cedex, France.
作者:
主题词
苯甲酰胺类(Benzamides);细胞遗传学分析(Cytogenetic Analysis);女(雌)性(Female);基因缺失(Gene Deletion);人类(Humans);原位杂交, 荧光(In Situ Hybridization, Fluorescence);核型分析(Karyotyping);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);男(雄)性(Male);哌嗪类(Piperazines);预后(Prognosis);蛋白激酶抑制剂(Protein Kinase Inhibitors);蛋白酪氨酸激酶类(Protein-Tyrosine Kinases);原癌基因蛋白质c-abl(Proto-Oncogene Proteins c-abl);原癌基因蛋白质c-bcr(Proto-Oncogene Proteins c-bcr);嘧啶类(Pyrimidines);回顾性研究(Retrospective Studies);存活率(Survival Rate);易位, 遗传(Translocation, Genetic)
DOI
10.1016/j.cancergencyto.2008.01.005
PMID
18406870
发布时间
2015-11-19
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