Mycoplasma infection suppresses p53, activates NF-kappaB and cooperates with oncogenic Ras in rodent fibroblast transformation.
第一作者:
D Y,Logunov
第一单位:
Gamaleya Research Institute for Epidemiology and Microbiology, Moscow, Russia.
作者:
医学主题词
动物(Animals);细胞凋亡(Apoptosis);细胞系(Cell Line);细胞转化, 肿瘤(Cell Transformation, Neoplastic);染色体不稳定性(Chromosomal Instability);周期素依赖激酶抑制剂p21(Cyclin-Dependent Kinase Inhibitor p21);DNA损伤(DNA Damage);胚胎, 哺乳动物(Embryo, Mammalian);成纤维细胞(Fibroblasts);人类(Humans);小鼠(Mice);支原体属(Mycoplasma);支原体感染(Mycoplasma Infections);NF-κB(NF-kappa B);原癌基因蛋白质类p21(ras)(Proto-Oncogene Proteins p21(ras));大鼠(Rats);反应元件(Response Elements);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53)
DOI
10.1038/onc.2008.103
PMID
18408766
发布时间
2021-10-20
基金项目
CA75179/CA/NCI NIH HHS/United States
R01 CA075179/CA/NCI NIH HHS/United States
CA098374/CA/NCI NIH HHS/United States
R01 CA098374/CA/NCI NIH HHS/United States
R01 CA060730/CA/NCI NIH HHS/United States
CA60730/CA/NCI NIH HHS/United States
- 浏览2
Oncogene
4521-31页
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