Overexpression of thioredoxin-1 reduces oxidative stress in the placenta of transgenic mice and promotes fetal growth via glucose metabolism.-论文-万方医学网

作者： Takashi,Umekawa ^[1] ; Takashi,Sugiyama ; Tomohisa,Kihira ; Nao,Murabayashi ; Lingyun,Zhang ; Kenji,Nagao ; Yuki,Kamimoto ; Ning,Ma ; Junji,Yodoi ; Norimasa,Sagawa

作者单位： Department of Obstetrics and Gynecology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie 514-8507, Japan. umkwtks@clin.medic.mie-u.ac.jp ^[1]

主题词动物(Animals);女(雌)性(Female);胎儿发育(Fetal Development);基因表达调控, 酶学(Gene Expression Regulation, Enzymologic);葡萄糖(Glucose);葡萄糖转运体1型(Glucose Transporter Type 1);人类(Humans);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);氧化还原(Oxidation-Reduction);氧化性应激(Oxidative Stress);胎盘(Placenta);胎盘形成(Placentation);妊娠(Pregnancy);RNA, 信使(RNA, Messenger);硫氧还蛋白质类(Thioredoxins);转染(Transfection);增量调节(Up-Regulation)

DOI 10.1210/en.2007-1682

PMID 18450962

发布时间 2024-11-30

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Overexpression of thioredoxin-1 reduces oxidative stress in the placenta of transgenic mice and promotes fetal growth via glucose metabolism.

Endocrinology

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Endocrinology

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Overexpression of thioredoxin-1 reduces oxidative stress in the placenta of transgenic mice and promotes fetal growth via glucose metabolism.

Endocrinology

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Endocrinology

相关期刊

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