换一批
换一批Toll-like receptor-4 modulates survival by induction of left ventricular remodeling after myocardial infarction in mice.
作者：
主题词
动物(Animals);心钠素(Atrial Natriuretic Factor);印迹法, 蛋白质(Blotting, Western);钙神经素(Calcineurin);胶原(Collagen);基因表达(Gene Expression);原位缺口末端标记(In Situ Nick-End Labeling);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);髓样分化因子88(Myeloid Differentiation Factor 88);心肌梗死(Myocardial Infarction);蛋白激酶Cδ(Protein Kinase C-delta);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);T淋巴细胞(T-Lymphocytes);Toll样受体4(Toll-Like Receptor 4);心室重构(Ventricular Remodeling)
DOI
10.4049/jimmunol.180.10.6954
PMID
18453617
发布时间
2022-03-11
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