Toll-like receptor-4 mediates neuronal apoptosis induced by amyloid beta-peptide and the membrane lipid peroxidation product 4-hydroxynonenal.
第一作者:
Sung-Chun,Tang
第一单位:
Laboratory of Neurosciences, National Institute on Aging Intramural Research Program, Baltimore, MD 21224, USA.
作者:
主题词
老年人(Aged);老年人, 80以上(Aged, 80 and over);醛类(Aldehydes);阿尔茨海默病(Alzheimer Disease);淀粉样β肽类(Amyloid beta-Peptides);动物(Animals);细胞凋亡(Apoptosis);脑(Brain);半胱氨酸天冬氨酸蛋白酶3(Caspase 3);女(雌)性(Female);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);脂质过氧化作用(Lipid Peroxidation);男(雄)性(Male);膜脂质类(Membrane Lipids);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);神经变性(Nerve Degeneration);氧化性应激(Oxidative Stress);肽碎片(Peptide Fragments);信号传导(Signal Transduction);Toll样受体4(Toll-Like Receptor 4)
DOI
10.1016/j.expneurol.2008.05.014
PMID
18586243
发布时间
2021-10-20
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Experimental neurology
114-21页
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