换一批Cryptic and partial deletions of PRDM16 and RUNX1 without t(1;21)(p36;q22) and/or RUNX1-PRDM16 fusion in a case of progressive chronic myeloid leukemia: a complex chromosomal rearrangement of underestimated frequency in disease progression?
第一作者：
Lauréline,Deluche
第一单位：
Cancer Research Institute of Lille, JP Aubert Center, Inserm Unit 837, Lille, France.
作者：
主题词
成年人(Adult);染色体, 人, 21对(Chromosomes, Human, Pair 21);核心结合因子α2亚基(Core Binding Factor Alpha 2 Subunit);DNA结合蛋白质类(DNA-Binding Proteins);疾病恶化(Disease Progression);女(雌)性(Female);基因复制(Gene Duplication);人类(Humans);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);模型, 遗传学(Models, Genetic);核酸杂交(Nucleic Acid Hybridization);癌基因蛋白质类, 融合(Oncogene Proteins, Fusion);蛋白质亚型(Protein Isoforms);重组融合蛋白质类(Recombinant Fusion Proteins);序列缺失(Sequence Deletion);转录因子(Transcription Factors);易位, 遗传(Translocation, Genetic);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1002/gcc.20611
PMID
18767145
发布时间
2008-10-13
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