Obesity and hypertension have differing oxidant handling molecular pathways in age-related chronic kidney disease.
第一作者:
C J,Percy
第一单位:
Molecular & Cellular Pathology, School of Medicine, The University of Queensland, Brisbane, Australia. c.percy@uq.edu.au
作者:
主题词
AMP活化蛋白激酶类(AMP-Activated Protein Kinases);体脂肪率(Adiposity);年龄因素(Age Factors);衰老(Aging);动物(Animals);自噬(Autophagy);体重(Body Weight);慢性病(Chronic Disease);疾病模型, 动物(Disease Models, Animal);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);纤维化(Fibrosis);叉头转录因子类(Forkhead Transcription Factors);血红素氧化酶(脱环)(Heme Oxygenase (Decyclizing));高血压(Hypertension);肾(Kidney);肾疾病(Kidney Diseases);L-乳酸脱氢酶(L-Lactate Dehydrogenase);男(雄)性(Male);NF-κB(NF-kappa B);神经组织蛋白质类(Nerve Tissue Proteins);肥胖症(Obesity);氧化剂(Oxidants);氧化性应激(Oxidative Stress);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);大鼠(Rats);大鼠, 近交SHR(Rats, Inbred SHR);大鼠, 近交WKY(Rats, Inbred WKY);Shc信号衔接蛋白质类(Shc Signaling Adaptor Proteins);信号传导(Signal Transduction);bcl-2相关X蛋白质(bcl-2-Associated X Protein)
DOI
10.1016/j.mad.2008.10.003
PMID
19041334
发布时间
2022-12-07
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