换一批
换一批Progressive aggregation despite chaperone associations of a mutant SOD1-YFP in transgenic mice that develop ALS.
第一作者：
Jiou,Wang
第一单位：
Howard Hughes Medical Institute, Department of Genetics, Section of Comparative Medicine, and Department of Neurosurgery, Yale University School of Medicine, New Haven, CT 06510, USA.
作者：
主题词
肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);神经胶质原纤维酸性蛋白质(Glial Fibrillary Acidic Protein);发光蛋白质类(Luminescent Proteins);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);分子伴侣(Molecular Chaperones);运动神经元(Motor Neurons);核酸杂交(Nucleic Acid Hybridization);溶解度(Solubility);脊髓(Spinal Cord);超氧化物歧化酶(Superoxide Dismutase);泛素(Ubiquitin)
DOI
10.1073/pnas.0813045106
PMID
19171884
发布时间
2022-03-09
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