Inducible knockout mutagenesis reveals compensatory mechanisms elicited by constitutive BK channel deficiency in overactive murine bladder.
作者:
主题词
动物(Animals);印迹法, 蛋白质(Blotting, Western);色谱法, 高压液相(Chromatography, High Pressure Liquid);环AMP(Cyclic AMP);免疫组织化学(Immunohistochemistry);大电导钙激活钾通道(Large-Conductance Calcium-Activated Potassium Channels);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肌收缩(Muscle Contraction);诱变(Mutagenesis);蛋白质组学(Proteomics);光谱法, 质量, 电喷雾电离(Spectrometry, Mass, Electrospray Ionization);串联质谱法(Tandem Mass Spectrometry);膀胱(Urinary Bladder);膀胱, 过度活动性(Urinary Bladder, Overactive)
DOI
10.1111/j.1742-4658.2009.06900.x
PMID
19220851
发布时间
2021-10-20
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The FEBS journal
1680-97页
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