换一批
换一批Noonan syndrome cardiac defects are caused by PTPN11 acting in endocardium to enhance endocardial-mesenchymal transformation.
第一作者：
Toshiyuki,Araki
第一单位：
Division of Hematology/Oncology and Cancer Biology Program, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02115, USA. taraki@uhnres.utoronto.c
作者：
主题词
等位基因(Alleles);动物(Animals);心内膜垫(Endocardial Cushions);心内膜(Endocardium);酶激活(Enzyme Activation);基因敲入技术(Gene Knock-In Techniques);心脏缺损, 先天性(Heart Defects, Congenital);心脏瓣膜(Heart Valves);中胚层(Mesoderm);小鼠(Mice);突变(Mutation);努南综合征(Noonan Syndrome);器官发生(Organogenesis);表型(Phenotype);蛋白质酪氨酸磷酸酶, 非受体11型(Protein Tyrosine Phosphatase, Non-Receptor Type 11);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt)
DOI
10.1073/pnas.0810053106
PMID
19251646
发布时间
2021-10-20
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