Activation of critical, host-induced, metabolic and stress pathways marks neutrophil entry into cystic fibrosis lungs.

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作者： Megha,Makam ^[1] ; Daisy,Diaz ; Julie,Laval ; Yael,Gernez ; Carol K,Conrad ; Colleen E,Dunn ; Zoe A,Davies ; Richard B,Moss ; Leonore A,Herzenberg ; Leonard A,Herzenberg ; Rabindra,Tirouvanziam

作者单位： Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA. ^[1]

主题词趋化性, 白细胞(Chemotaxis, Leukocyte);cAMP反应元件结合蛋白质(Cyclic AMP Response Element-Binding Protein);囊性纤维化(Cystic Fibrosis);人类(Humans);肺疾病(Lung Diseases);中性白细胞(Neutrophils);氧化性应激(Oxidative Stress);磷酰化(Phosphorylation);蛋白激酶类(Protein Kinases);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases)

DOI 10.1073/pnas.0813410106

PMID 19293384

发布时间 2021-12-03

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Proceedings of the National Academy of Sciences of the United States of America

2009年106卷14期

5779-83页

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Activation of critical, host-induced, metabolic and stress pathways marks neutrophil entry into cystic fibrosis lungs.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

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Activation of critical, host-induced, metabolic and stress pathways marks neutrophil entry into cystic fibrosis lungs.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

相关期刊

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