Major urinary protein-1 increases energy expenditure and improves glucose intolerance through enhancing mitochondrial function in skeletal muscle of diabetic mice.
作者:
主题词
动物(Animals);糖尿病, 实验性(Diabetes Mellitus, Experimental);膳食脂肪类(Dietary Fats);能量代谢(Energy Metabolism);摄食行为(Feeding Behavior);葡糖耐受不良(Glucose Intolerance);胰岛素(Insulin);胰岛素抗药性(Insulin Resistance);脂类代谢(Lipid Metabolism);肝(Liver);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 肥胖(Mice, Obese);线粒体(Mitochondria);运动活动(Motor Activity);肌, 骨骼(Muscle, Skeletal);蛋白质类(Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);重组蛋白质类(Recombinant Proteins);信号传导(Signal Transduction);噻唑烷二酮类(Thiazolidinediones);增量调节(Up-Regulation)
DOI
10.1074/jbc.M109.001107
PMID
19336396
发布时间
2024-04-16
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