G-CSF-initiated myeloid cell mobilization and angiogenesis mediate tumor refractoriness to anti-VEGF therapy in mouse models.
第一作者:
Farbod,Shojaei
第一单位:
Genentech, Inc., 1 DNA Way, South San Francisco, CA 94080, USA.
作者:
医学主题词
动物(Animals);抗体, 肿瘤(Antibodies, Neoplasm);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);抗药性, 肿瘤(Drug Resistance, Neoplasm);药物筛选试验, 抗肿瘤(Drug Screening Assays, Antitumor);胃肠激素类(Gastrointestinal Hormones);粒细胞集落刺激因子(Granulocyte Colony-Stimulating Factor);人类(Humans);小鼠(Mice);小鼠, 裸(Mice, Nude);髓系细胞(Myeloid Cells);肿瘤(Neoplasms);新生血管化, 病理性(Neovascularization, Pathologic);神经肽类(Neuropeptides);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1073/pnas.0902280106
PMID
19346489
发布时间
2021-10-20
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