换一批
换一批Increased AKT S473 phosphorylation after mTORC1 inhibition is rictor dependent and does not predict tumor cell response to PI3K/mTOR inhibition.
作者：
主题词
载体蛋白质类(Carrier Proteins);细胞增殖(Cell Proliferation);细胞存活(Cell Survival);人类(Humans);咪唑类(Imidazoles);免疫印迹法(Immunoblotting);免疫抑制剂(Immunosuppressive Agents);胰岛素受体底物蛋白质类(Insulin Receptor Substrate Proteins);多蛋白复合物(Multiprotein Complexes);肿瘤(Neoplasms);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);蛋白激酶类(Protein Kinases);蛋白质类(Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);喹啉类(Quinolines);RNA, 小分子干扰(RNA, Small Interfering);西罗莫司(Sirolimus);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);转录因子(Transcription Factors);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1158/1535-7163.MCT-08-0668
PMID
19372546
发布时间
2023-11-05
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