换一批Loss of Nkx3.1 leads to the activation of discrete downstream target genes during prostate tumorigenesis.
第一作者：
H,Song
第一单位：
Department of Pathology & Immunology, School of Medicine, Washington University at St Louis, St Louis, MO 63110, USA.
作者：
医学主题词
动物(Animals);萎缩(Atrophy);聚集素(Clusterin);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);减量调节(Down-Regulation);基因缺失(Gene Deletion);基因表达谱(Gene Expression Profiling);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);同源盒结构域蛋白质类(Homeodomain Proteins);人类(Humans);激光(Lasers);男(雄)性(Male);小鼠(Mice);显微切割(Microdissection);作用于硫族供体的氧化还原酶类(Oxidoreductases Acting on Sulfur Group Donors);PTEN磷酸水解酶(PTEN Phosphohydrolase);前列腺(Prostate);前列腺肿瘤(Prostatic Neoplasms);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);原癌基因蛋白质c-myc(Proto-Oncogene Proteins c-myc);信号传导(Signal Transduction);硫氧还蛋白质类(Thioredoxins);转录因子(Transcription Factors);转录, 遗传(Transcription, Genetic);转录激活(Transcriptional Activation)
DOI
10.1038/onc.2009.181
PMID
19597465
发布时间
2025-05-29
- 浏览22
Oncogene
3307-19页
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