Maternal obesity accelerates fetal pancreatic beta-cell but not alpha-cell development in sheep: prenatal consequences.
第一作者:
Stephen P,Ford
第一单位:
Center for the Study of Fetal Programming, Department of Animal Science, University of Wyoming, Laramie, Wyoming, USA.
作者:
主题词
动物(Animals);动物, 新生(Animals, Newborn);血糖(Blood Glucose);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);胎儿发育(Fetal Development);胎儿(Fetus);孕龄(Gestational Age);胰高血糖素分泌细胞(Glucagon-Secreting Cells);氢化可的松(Hydrocortisone);胰岛素(Insulin);胰岛素抗药性(Insulin Resistance);胰岛素分泌细胞(Insulin-Secreting Cells);母亲营养生理学现象(Maternal Nutritional Physiological Phenomena);有丝分裂(Mitosis);肥胖症(Obesity);胰腺(Pancreas);妊娠(Pregnancy);产前暴露迟发效应(Prenatal Exposure Delayed Effects);绵羊(Sheep);增量调节(Up-Regulation)
DOI
10.1152/ajpregu.00072.2009
PMID
19605766
发布时间
2021-10-20
- 浏览6

相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文