换一批
换一批Understanding the molecular mechanism of blood-brain barrier damage in an experimental model of Japanese encephalitis: correlation with minocycline administration as a therapeutic agent.
作者：
主题词
动物(Animals);抗菌药(Anti-Bacterial Agents);血脑屏障(Blood-Brain Barrier);细胞黏附分子(Cell Adhesion Molecules);趋化性, 白细胞(Chemotaxis, Leukocyte);着色剂(Coloring Agents);环氧化酶2(Cyclooxygenase 2);疾病模型, 动物(Disease Models, Animal);脑炎病毒, 日本(Encephalitis Virus, Japanese);脑炎, 日本(Encephalitis, Japanese);伊文思蓝(Evans Blue);女(雌)性(Female);男(雄)性(Male);基质金属蛋白酶9(Matrix Metalloproteinase 9);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);米诺环素(Minocycline);神经保护药(Neuroprotective Agents);一氧化氮合酶Ⅱ型(Nitric Oxide Synthase Type II);RNA, 信使(RNA, Messenger);受体, 趋化因子(Receptors, Chemokine);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1016/j.neuint.2009.07.006
PMID
19628016
发布时间
2022-03-21
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