Adaptive mutations in a human immunodeficiency virus type 1 envelope protein with a truncated V3 loop restore function by improving interactions with CD4.

导出 OA

翻译打印收藏纠错

作者： Caroline,Agrawal-Gamse ^[1] ; Fang-Hua,Lee ; Beth,Haggarty ; Andrea P O,Jordan ; Yanjie,Yi ; Benhur,Lee ; Ronald G,Collman ; James A,Hoxie ; Robert W,Doms ; Meg M,Laakso

作者单位： Department of Microbiology, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA. ^[1]

主题词适应, 生物学(Adaptation, Biological);动物(Animals);抗HIV药(Anti-HIV Agents);抗体, 单克隆(Antibodies, Monoclonal);细胞系(Cell Line);环己烷类(Cyclohexanes);HIV包膜蛋白质gp120(HIV Envelope Protein gp120);HIV-1(HIV-1);人类(Humans);突变(Mutation);受体, CCR5(Receptors, CCR5);重组蛋白质类(Recombinant Proteins);三唑类(Triazoles)

DOI 10.1128/JVI.01238-09

PMID 19692476

发布时间 2021-10-20

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览68

Journal of virology

2009年83卷21期

11005-15页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Adaptive mutations in a human immunodeficiency virus type 1 envelope protein with a truncated V3 loop restore function by improving interactions with CD4.

Journal of virology

相似文献

Journal of virology

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Adaptive mutations in a human immunodeficiency virus type 1 envelope protein with a truncated V3 loop restore function by improving interactions with CD4.

Journal of virology

相似文献

Journal of virology

相关期刊

检索历史清除