换一批
换一批The nuclear receptor PPAR gamma selectively inhibits Th17 differentiation in a T cell-intrinsic fashion and suppresses CNS autoimmunity.
第一作者：
Luisa,Klotz
第一单位：
Institutes of Molecular Medicine and Experimental Immunology, Department of Neurology, University of Bonn, Bonn 53105, Germany.
作者：
主题词
动物(Animals);细胞分化(Cell Differentiation);DNA结合蛋白质类(DNA-Binding Proteins);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);人类(Humans);白细胞介素17(Interleukin-17);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);多发性硬化(Multiple Sclerosis);核受体共阻遏蛋白2(Nuclear Receptor Co-Repressor 2);核受体亚家族1, F组, 成员3(Nuclear Receptor Subfamily 1, Group F, Member 3);PPARγ(PPAR gamma);启动区, 遗传(Promoter Regions, Genetic);受体, 维甲酸(Receptors, Retinoic Acid);受体, 甲状腺激素(Receptors, Thyroid Hormone);阻遏蛋白质类(Repressor Proteins);T淋巴细胞, 辅助诱导(T-Lymphocytes, Helper-Inducer)
DOI
10.1084/jem.20082771
PMID
19737866
发布时间
2021-10-20
- 浏览36
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
