Myoglobin-deficient mice activate a distinct cardiac gene expression program in response to isoproterenol-induced hypertrophy.
作者:
主题词
适应, 生理学(Adaptation, Physiological);动物(Animals);聚类分析(Cluster Analysis);疾病模型, 动物(Disease Models, Animal);基因表达谱(Gene Expression Profiling);基因表达调控(Gene Expression Regulation);基因调控网络(Gene Regulatory Networks);基因型(Genotype);肥大, 左心室(Hypertrophy, Left Ventricular);异丙肾上腺素(Isoproterenol);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);心肌(Myocardium);肌红蛋白(Myoglobin);寡核苷酸序列分析(Oligonucleotide Array Sequence Analysis);表型(Phenotype);每搏输出量(Stroke Volume);时间因素(Time Factors);超声检查(Ultrasonography);心室功能, 左(Ventricular Function, Left);心室压(Ventricular Pressure);心室重构(Ventricular Remodeling)
DOI
10.1152/physiolgenomics.90297.2008
PMID
20145201
发布时间
2016-11-25
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Physiological genomics
137-45页
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