T and B cell hyperactivity and autoimmunity associated with niche-specific defects in apoptotic body clearance in TIM-4-deficient mice.
第一单位:
Department of Neurology, Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Harvard Institutes of Medicine, Boston, MA 02115, USA.
作者:
主题词
动物(Animals);抗体, 抗核(Antibodies, Antinuclear);抗原呈递细胞(Antigen-Presenting Cells);细胞凋亡(Apoptosis);自身免疫(Autoimmunity);B-淋巴细胞(B-Lymphocytes);细胞区室化(Cell Compartmentation);女(雌)性(Female);淋巴细胞活化(Lymphocyte Activation);巨噬细胞, 腹膜(Macrophages, Peritoneal);男(雄)性(Male);膜蛋白质类(Membrane Proteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);吞噬作用(Phagocytosis);表型(Phenotype);重组, 遗传(Recombination, Genetic);脾(Spleen);T淋巴细胞(T-Lymphocytes);胸腺(Thymus Gland)
DOI
10.1073/pnas.0910359107
PMID
20368430
发布时间
2022-03-10
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