Endoplasmic reticulum accumulation of Kir6.2 without activation of ER stress response in islet cells from adult Sur1 knockout mice.
第一作者:
Ihsane,Marhfour
第一单位:
Department of Pathology, Cliniques Universitaires Saint-Luc, Université Catholique de Louvain, Tour Rosalind Franklin, 10 Avenue Hippocrate, 1200, Brussels, Belgium.
作者:
医学主题词
ATP结合匣式转运子(ATP-Binding Cassette Transporters);动物(Animals);抗体特异性(Antibody Specificity);钙网蛋白(Calreticulin);内质网(Endoplasmic Reticulum);基因表达调控(Gene Expression Regulation);胰岛(Islets of Langerhans);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);氧化性应激(Oxidative Stress);钾通道, 内向整流(Potassium Channels, Inwardly Rectifying);蛋白质转运(Protein Transport);RNA, 信使(RNA, Messenger);受体, 药物(Receptors, Drug);应激, 生理学(Stress, Physiological);亚细胞部分(Subcellular Fractions)
DOI
10.1007/s00441-010-0958-8
PMID
20383647
发布时间
2017-12-13
- 浏览14
Cell and tissue research
335-46页
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