Cellular localization of the herpes simplex virus ICP0 protein dictates its ability to block IRF3-mediated innate immune responses.

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作者： Patrick,Paladino ^[1] ; Susan E,Collins ; Karen L,Mossman

作者单位： Department of Pathology and Molecular Medicine, Institute for Infectious Disease Research, McMaster University, Hamilton, Ontario, Canada. ^[1]

主题词主动转运, 细胞核(Active Transport, Cell Nucleus);细胞, 培养的(Cells, Cultured);细胞质(Cytoplasm);单纯疱疹(Herpes Simplex);疱疹病毒1型, 人(Herpesvirus 1, Human);人类(Humans);即早蛋白质类(Immediate-Early Proteins);免疫, 先天(Immunity, Innate);干扰素调节因子3(Interferon Regulatory Factor-3);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);病毒蛋白质类(Viral Proteins)

DOI 10.1371/journal.pone.0010428

PMID 20454685

发布时间 2021-10-20

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PloS one

2010年5卷4期

e10428页

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Cellular localization of the herpes simplex virus ICP0 protein dictates its ability to block IRF3-mediated innate immune responses.

PloS one

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Cellular localization of the herpes simplex virus ICP0 protein dictates its ability to block IRF3-mediated innate immune responses.

PloS one

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PloS one

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