Densities of glutamatergic and GABAergic presynaptic terminals are altered in experimental cortical dysplasia.
第一作者:
Fu-Wen,Zhou
第一单位:
Department of Neurosurgery and the McKnight Brain Institute, University of Florida, Gainesville, Florida, USA.
作者:
主题词
方差分析(Analysis of Variance);动物(Animals);动物, 新生(Animals, Newborn);大脑皮质(Cerebral Cortex);疾病模型, 动物(Disease Models, Animal);胚胎, 哺乳动物(Embryo, Mammalian);女(雌)性(Female);谷氨酸(Glutamic Acid);半身照射(Hemibody Irradiation);男(雄)性(Male);皮质发育畸形(Malformations of Cortical Development);小白蛋白(Parvalbumins);妊娠(Pregnancy);产前暴露迟发效应(Prenatal Exposure Delayed Effects);突触前末梢(Presynaptic Terminals);大鼠(Rats);囊泡谷氨酸转运蛋白质2(Vesicular Glutamate Transport Protein 2);囊泡谷氨酸转运蛋白质类(Vesicular Glutamate Transport Proteins);囊泡抑制性氨基酸转运蛋白质类(Vesicular Inhibitory Amino Acid Transport Proteins);γ氨基丁酸(gamma-Aminobutyric Acid)
DOI
10.1111/j.1528-1167.2010.02583.x
PMID
20477846
发布时间
2020-10-21
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Epilepsia
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