Program death-1 signaling and regulatory T cells collaborate to resist the function of adoptively transferred cytotoxic T lymphocytes in advanced acute myeloid leukemia.
第一作者:
Qing,Zhou
第一单位:
Masonic Cancer Center and Department of Pediatrics, Division of Blood and Marrow Transplantation, University of Minnesota, Minneapolis, MN 55455, USA.
作者:
医学主题词
动物(Animals);抗体, 单克隆(Antibodies, Monoclonal);抗原, 表面(Antigens, Surface);凋亡调节蛋白质类(Apoptosis Regulatory Proteins);基因表达调控, 白血病(Gene Expression Regulation, Leukemic);白血病, 髓样, 急性(Leukemia, Myeloid, Acute);淋巴细胞去除术(Lymphocyte Depletion);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);肽类(Peptides);T淋巴细胞, 细胞毒性(T-Lymphocytes, Cytotoxic);T淋巴细胞, 调节性(T-Lymphocytes, Regulatory)
DOI
10.1182/blood-2010-03-275446
PMID
20570856
发布时间
2022-03-11
基金项目
R01AI34495/AI/NIAID NIH HHS/United States
P01 AI056299/AI/NIAID NIH HHS/United States
P01056299/PHS HHS/United States
R01 AI034495/AI/NIAID NIH HHS/United States
R01 CA72669/CA/NCI NIH HHS/United States
R01 HL056067/HL/NHLBI NIH HHS/United States
R01 CA072669/CA/NCI NIH HHS/United States
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Blood
2484-93页
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