换一批
换一批An increase in spinal dehydroepiandrosterone sulfate (DHEAS) enhances NMDA-induced pain via phosphorylation of the NR1 subunit in mice: involvement of the sigma-1 receptor.
第一作者：
Seo-Yeon,Yoon
第一单位：
Department of Anesthesiology and Pain Medicine, University of Texas MD Anderson Cancer Center, Houston, TX, USA.
作者：
主题词
方差分析(Analysis of Variance);动物(Animals);环AMP依赖性蛋白激酶类(Cyclic AMP-Dependent Protein Kinases);硫酸脱氢表雄酮(Dehydroepiandrosterone Sulfate);免疫组织化学(Immunohistochemistry);注射, 脊髓(Injections, Spinal);男(雄)性(Male);小鼠(Mice);小鼠, 近交ICR(Mice, Inbred ICR);N-甲基天冬氨酸(N-Methylaspartate);疼痛(Pain);磷酰化(Phosphorylation);蛋白激酶C(Protein Kinase C);受体, GABA-A(Receptors, GABA-A);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);受体, Σ(Receptors, sigma);脊髓(Spinal Cord)
DOI
10.1016/j.neuropharm.2010.06.007
PMID
20600171
发布时间
2023-12-13
- 浏览16
Neuropharmacology
460-7页
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