Salt-induced cardiac hypertrophy and interstitial fibrosis are due to a blood pressure-independent mechanism in Wistar rats.
第一作者:
Daniele N,Ferreira
第一单位:
Laboratory of Experimental Hypertension, University of São Paulo, São Paulo 01246-903, Brazil.
作者:
主题词
醛固酮(Aldosterone);血管紧张素Ⅱ(Angiotensin II);血管紧张素Ⅱ1型受体拮抗剂(Angiotensin II Type 1 Receptor Blockers);动物(Animals);抗高血压药(Antihypertensive Agents);血压(Blood Pressure);心脏扩大(Cardiomegaly);胶原Ⅰ型(Collagen Type I);胶原Ⅲ型(Collagen Type III);疾病模型, 动物(Disease Models, Animal);饮水(Drinking);进食(Eating);超声心动描记术(Echocardiography);纤维化(Fibrosis);基因表达(Gene Expression);心室(Heart Ventricles);肼苯哒嗪(Hydralazine);高血压(Hypertension);洛沙坦(Losartan);男(雄)性(Male);心肌(Myocardium);钾(Potassium);大鼠(Rats);大鼠, Wistar(Rats, Wistar);受体, 血管紧张素, 1型(Receptor, Angiotensin, Type 1);受体, 血管紧张素, 2型(Receptor, Angiotensin, Type 2);肾素(Renin);肾素-血管紧张素系统(Renin-Angiotensin System);钠(Sodium);氯化钠, 膳食(Sodium Chloride, Dietary);转化生长因子β(Transforming Growth Factor beta);尿(Urine)
DOI
10.3945/jn.109.117473
PMID
20724490
发布时间
2023-02-16
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The Journal of nutrition
1742-51页
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