Cytomegalovirus infection induces the accumulation of short-lived, multifunctional CD4+CD45RA+CD27+ T cells: the potential involvement of interleukin-7 in this process.

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作者： Valentina,Libri ^[1] ; Rita I,Azevedo ; Sarah E,Jackson ; Diletta,Di Mitri ; Raskit,Lachmann ; Stephan,Fuhrmann ; Milica,Vukmanovic-Stejic ; Kwee,Yong ; Luca,Battistini ; Florian,Kern ; Maria V D,Soares ; Arne N,Akbar

作者单位： Division of Infection and Immunity, University College London, London, UK. ^[1]

主题词成年人(Adult);老年人(Aged);老年人, 80以上(Aged, 80 and over);衰老(Aging);CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);细胞分化(Cell Differentiation);细胞分离(Cell Separation);细胞存活(Cell Survival);巨细胞病毒感染(Cytomegalovirus Infections);流式细胞术(Flow Cytometry);人类(Humans);白细胞介素7(Interleukin-7);中年人(Middle Aged);信号传导(Signal Transduction);T淋巴细胞亚群(T-Lymphocyte Subsets);青年人(Young Adult)

DOI 10.1111/j.1365-2567.2010.03386.x

PMID 21214539

发布时间 2022-03-11

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Immunology

2011年132卷3期

326-39页

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Cytomegalovirus infection induces the accumulation of short-lived, multifunctional CD4+CD45RA+CD27+ T cells: the potential involvement of interleukin-7 in this process.

Immunology

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Immunology

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Cytomegalovirus infection induces the accumulation of short-lived, multifunctional CD4+CD45RA+CD27+ T cells: the potential involvement of interleukin-7 in this process.

Immunology

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Immunology

相关期刊

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