换一批Overexpression of CaMKIIδc in RyR2R4496C+/- knock-in mice leads to altered intracellular Ca2+ handling and increased mortality.
第一作者：
Nataliya,Dybkova
第一单位：
Department of Cardiology and Pneumology, Georg-August-University Göttingen, Göttingen, Germany.
作者：
医学主题词
方差分析(Analysis of Variance);动物(Animals);心律失常, 心性(Arrhythmias, Cardiac);钙(Calcium);钙信号(Calcium Signaling);钙-钙调素依赖性蛋白激酶2型(Calcium-Calmodulin-Dependent Protein Kinase Type 2);心脏扩大(Cardiomegaly);卡方分布(Chi-Square Distribution);疾病模型, 动物(Disease Models, Animal);超声心动描记术(Echocardiography);兴奋收缩偶联(Excitation Contraction Coupling);基因表达调控(Gene Expression Regulation);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);小鼠, 转基因(Mice, Transgenic);心肌收缩(Myocardial Contraction);磷酰化(Phosphorylation);随机分配(Random Allocation);兰尼碱受体钙释放通道(Ryanodine Receptor Calcium Release Channel)
DOI
10.1016/j.jacc.2010.08.639
PMID
21251589
发布时间
2025-05-29
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