换一批CC chemokine receptor 5 deletion impairs macrophage activation and induces adverse remodeling following myocardial infarction.
第一作者：
Rogelio,Zamilpa
第一单位：
Division of Geriatrics, Gerontology and Palliative Medicine, Department of Medicine, The University of Texas Health Science Center at San Antonio, USA.
作者：
主题词
动物(Animals);抗原, CD(Antigens, CD);抗原, 分化, 髓单核细胞(Antigens, Differentiation, Myelomonocytic);精氨酸酶(Arginase);胶原Ⅰ型(Collagen Type I);女(雌)性(Female);基因缺失(Gene Deletion);HSP47热休克蛋白质类(HSP47 Heat-Shock Proteins);白细胞介素1β(Interleukin-1beta);白细胞介素6(Interleukin-6);外源凝集素类, C型(Lectins, C-Type);巨噬细胞活化(Macrophage Activation);巨噬细胞(Macrophages);男(雄)性(Male);甘露糖结合凝集素类(Mannose-Binding Lectins);小鼠(Mice);心肌梗死(Myocardial Infarction);前胶原(Procollagen);受体, CCR5(Receptors, CCR5);受体, 细胞表面(Receptors, Cell Surface);转化生长因子β1(Transforming Growth Factor beta1);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);心室重构(Ventricular Remodeling)
DOI
10.1152/ajpheart.01002.2010
PMID
21297029
发布时间
2025-01-03
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