Nonreceptor tyrosine kinase BMX maintains self-renewal and tumorigenic potential of glioblastoma stem cells by activating STAT3.
第一作者:
Olga A,Guryanova
第一单位:
Department of Stem Cell Biology and Regenerative Medicine, Lerner Research Institute, Cleveland Clinic, Cleveland, OH 44195, USA.
作者:
医学主题词
动物(Animals);碱性螺旋-环-螺旋转录因子类(Basic Helix-Loop-Helix Transcription Factors);脑肿瘤(Brain Neoplasms);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);胶质母细胞瘤(Glioblastoma);人类(Humans);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);肿瘤干细胞(Neoplastic Stem Cells);神经组织蛋白质类(Nerve Tissue Proteins);蛋白酪氨酸激酶类(Protein-Tyrosine Kinases);受体, 白细胞介素6(Receptors, Interleukin-6);STAT3转录因子(STAT3 Transcription Factor);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1016/j.ccr.2011.03.004
PMID
21481791
发布时间
2025-05-29
- 浏览139
Cancer cell
498-511页
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