Serum and liver iron differently regulate the bone morphogenetic protein 6 (BMP6)-SMAD signaling pathway in mice.
第一作者:
Elena,Corradini
第一单位:
Program in Membrane Biology, Division of Nephrology, Center for Systems Biology, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.
作者:
主题词
动物(Animals);抗微生物阳离子肽类(Antimicrobial Cationic Peptides);骨形态发生蛋白质6(Bone Morphogenetic Protein 6);内环境稳定(Homeostasis);铁(Iron);肝(Liver);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);模型, 动物(Models, Animal);信号传导(Signal Transduction);Smad蛋白质类(Smad Proteins);转铁蛋白(Transferrin)
DOI
10.1002/hep.24359
PMID
21488083
发布时间
2021-10-20
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