Aryl hydrocarbon receptor-induced signals up-regulate IL-22 production and inhibit inflammation in the gastrointestinal tract.
作者:
主题词
成年人(Adult);动物(Animals);碱性螺旋-环-螺旋转录因子类(Basic Helix-Loop-Helix Transcription Factors);活组织检查(Biopsy);卡唑类(Carbazoles);病例对照研究(Case-Control Studies);细胞, 培养的(Cells, Cultured);硫酸葡聚糖(Dextran Sulfate);疾病模型, 动物(Disease Models, Animal);剂量效应关系, 药物(Dose-Response Relationship, Drug);女(雌)性(Female);流式细胞术(Flow Cytometry);人类(Humans);炎症介导素类(Inflammation Mediators);炎性肠疾病(Inflammatory Bowel Diseases);白细胞介素类(Interleukins);肠(Intestines);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);中年人(Middle Aged);聚合酶链反应(Polymerase Chain Reaction);受体, 芳基烃(Receptors, Aryl Hydrocarbon);疾病严重程度指数(Severity of Illness Index);信号传导(Signal Transduction);T淋巴细胞(T-Lymphocytes);时间因素(Time Factors);三硝基苯磺酸(Trinitrobenzenesulfonic Acid);增量调节(Up-Regulation)
DOI
10.1053/j.gastro.2011.04.007
PMID
21600206
发布时间
2023-12-13
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Gastroenterology
237-48, 248.e1页
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