换一批
换一批Liver-specific deletion of protein tyrosine phosphatase (PTP) 1B improves obesity- and pharmacologically induced endoplasmic reticulum stress.
第一作者：
Abdelali,Agouni
第一单位：
Institute of Biological and Environmental Sciences, College of Life Sciences and Medicine, University of Aberdeen, UK.
作者：
主题词
激活转录因子6(Activating Transcription Factor 6);动物(Animals);细胞核(Cell Nucleus);Ia类磷脂酰肌醇3-激酶(Class Ia Phosphatidylinositol 3-Kinase);DNA结合蛋白质类(DNA-Binding Proteins);内质网(Endoplasmic Reticulum);内切核糖核酸酶类(Endoribonucleases);真核细胞起始因子2(Eukaryotic Initiation Factor-2);基因缺失(Gene Deletion);基因敲低技术(Gene Knockdown Techniques);葡萄糖(Glucose);Hep G2细胞(Hep G2 Cells);内环境稳定(Homeostasis);人类(Humans);脂类代谢(Lipid Metabolism);肝(Liver);小鼠(Mice);肥胖症(Obesity);器官特异性(Organ Specificity);蛋白质转运(Protein Transport);蛋白质酪氨酸磷酸酶, 非受体1型(Protein Tyrosine Phosphatase, Non-Receptor Type 1);RNA, 信使(RNA, Messenger);信号传导(Signal Transduction);应激, 生理学(Stress, Physiological);毒胡萝卜内酯(Thapsigargin);转录因子(Transcription Factors);衣霉素(Tunicamycin)
DOI
10.1042/BJ20110373
PMID
21605081
发布时间
2024-01-09
- 浏览48
The Biochemical journal
369-78页
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