The 894G > T (Glu298Asp) variant in the endothelial NOS gene and MTHFR polymorphisms influence homocysteine levels in patients with cognitive decline.

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作者： Nadia,Ferlazzo ^[1] ; Gaetano,Gorgone ; Daniela,Caccamo ; Monica,Currò ; Salvatore,Condello ; Francesco,Pisani ; Fabrizio,Vernieri ; Paolo Maria,Rossini ; Riccardo,Ientile

作者单位： Department of Biochemical, Physiological and Nutritional Sciences, University of Messina, Messina, Italy. ^[1]

主题词老年人(Aged);老年人, 80以上(Aged, 80 and over);阿尔茨海默病(Alzheimer Disease);认知障碍(Cognition Disorders);女(雌)性(Female);基因型(Genotype);人类(Humans);高同种半胱氨酸血症(Hyperhomocysteinemia);男(雄)性(Male);亚甲基四氢叶酸还原酶(NADPH)(Methylenetetrahydrofolate Reductase (NADPH2));一氧化氮合酶Ⅲ型(Nitric Oxide Synthase Type III);多态现象, 遗传(Polymorphism, Genetic);危险因素(Risk Factors)

DOI 10.1007/s12017-011-8148-8

PMID 21607713

发布时间 2021-10-20

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Neuromolecular medicine

2011年13卷3期

167-74页

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The 894G > T (Glu298Asp) variant in the endothelial NOS gene and MTHFR polymorphisms influence homocysteine levels in patients with cognitive decline.

Neuromolecular medicine

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Neuromolecular medicine

相关期刊

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The 894G &gt; T (Glu298Asp) variant in the endothelial NOS gene and MTHFR polymorphisms influence homocysteine levels in patients with cognitive decline.

Neuromolecular medicine

相似文献

Neuromolecular medicine

相关期刊

检索历史清除

The 894G > T (Glu298Asp) variant in the endothelial NOS gene and MTHFR polymorphisms influence homocysteine levels in patients with cognitive decline.