Altered composition of fatty acids exacerbates hepatotumorigenesis during activation of the phosphatidylinositol 3-kinase pathway.
第一作者:
Yotaro,Kudo
第一单位:
Department of Gastroenterology, Graduate School of Medicine, The University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-8655, Japan.
作者:
医学主题词
动物(Animals);碱基序列(Base Sequence);I类磷脂酰肌醇3-激酶类(Class I Phosphatidylinositol 3-Kinases);DNA引物(DNA Primers);减量调节(Down-Regulation);酶激活(Enzyme Activation);脂肪酸类(Fatty Acids);脂肪肝(Fatty Liver);基因表达(Gene Expression);基因, 肿瘤抑制(Genes, Tumor Suppressor);肝细胞(Hepatocytes);肝(Liver);肝肿瘤, 实验性(Liver Neoplasms, Experimental);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);突变蛋白质类(Mutant Proteins);油酸类(Oleic Acids);棕榈酸类(Palmitic Acids);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);重组蛋白质类(Recombinant Proteins);信号传导(Signal Transduction)
DOI
10.1016/j.jhep.2011.03.025
PMID
21703185
发布时间
2020-12-09
- 浏览12
Journal of hepatology
1400-8页
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