Beneficial effects of ethyl pyruvate through inhibiting high-mobility group box 1 expression and TLR4/NF-κB pathway after traumatic brain injury in the rat.

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作者： Xingfen,Su ^[1] ; Handong,Wang ; Jinbing,Zhao ; Hao,Pan ; Lei,Mao

作者单位： Department of Neurosurgery, Jinling Hospital, School of Medicine, Nanjing University, 305 East Zhongshan Road, Jiangsu Province, Nanjing 210002, China. ^[1]

主题词动物(Animals);消炎药(Anti-Inflammatory Agents);脑水肿(Brain Edema);脑损伤(Brain Injuries);HMGB1蛋白质(HMGB1 Protein);原位缺口末端标记(In Situ Nick-End Labeling);男(雄)性(Male);NF-κB(NF-kappa B);神经保护药(Neuroprotective Agents);神经心理学测验(Neuropsychological Tests);丙酮酸盐类(Pyruvates);随机分配(Random Allocation);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley)

DOI 10.1155/2011/807142

PMID 21772666

发布时间 2021-10-20

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Mediators of inflammation

2011年2011卷

807142页

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Beneficial effects of ethyl pyruvate through inhibiting high-mobility group box 1 expression and TLR4/NF-κB pathway after traumatic brain injury in the rat.

Mediators of inflammation

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Mediators of inflammation

相关期刊

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Beneficial effects of ethyl pyruvate through inhibiting high-mobility group box 1 expression and TLR4/NF-κB pathway after traumatic brain injury in the rat.

Mediators of inflammation

相似文献

Mediators of inflammation

相关期刊

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