Endogenous neurotrophins and Trk signaling in diffuse large B cell lymphoma cell lines are involved in sensitivity to rituximab-induced apoptosis.
第一作者:
Cynthia,Bellanger
第一单位:
Université de Limoges, EA3842, Limoges, France.
作者:
主题词
抗体, 单克隆, 鼠源性(Antibodies, Monoclonal, Murine-Derived);抗肿瘤药(Antineoplastic Agents);印迹法, 蛋白质(Blotting, Western);脑源性神经营养因子(Brain-Derived Neurotrophic Factor);抗药性, 肿瘤(Drug Resistance, Neoplasm);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);流式细胞术(Flow Cytometry);荧光抗体技术(Fluorescent Antibody Technique);人类(Humans);免疫酶技术(Immunoenzyme Techniques);淋巴瘤, B细胞(Lymphoma, B-Cell);淋巴瘤, 大B细胞, 弥漫性(Lymphoma, Large B-Cell, Diffuse);神经生长因子(Nerve Growth Factor);神经生长因子类(Nerve Growth Factors);RNA, 信使(RNA, Messenger);受体, 神经生长因子(Receptor, Nerve Growth Factor);受体, trkA(Receptor, trkA);受体, trkB(Receptor, trkB);受体, trkC(Receptor, trkC);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1371/journal.pone.0027213
PMID
22076137
发布时间
2021-10-21
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PloS one
e27213页
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