Glucocorticoid-induced leucine zipper (GILZ) over-expression in T lymphocytes inhibits inflammation and tissue damage in spinal cord injury.
第一作者:
Emanuela,Esposito
第一单位:
Department of Clinical and Experimental Medicine and Pharmacology, School of Medicine, University of Messina, Torre Biologica, Policlinico Universitario, Messina, Italy.
作者:
主题词
动物(Animals);细胞凋亡(Apoptosis);细胞周期蛋白D1(Cyclin D1);细胞因子类(Cytokines);地诺前列酮(Dinoprostone);疾病模型, 动物(Disease Models, Animal);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);基因表达调控(Gene Expression Regulation);神经胶质原纤维酸性蛋白质(Glial Fibrillary Acidic Protein);原位缺口末端标记(In Situ Nick-End Labeling);炎症(Inflammation);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);一氧化氮合酶Ⅱ型(Nitric Oxide Synthase Type II);过氧化物酶(Peroxidase);信号传导(Signal Transduction);脊髓损伤(Spinal Cord Injuries);T淋巴细胞(T-Lymphocytes);转录因子(Transcription Factors);bcl-2相关X蛋白质(bcl-2-Associated X Protein)
DOI
10.1007/s13311-011-0084-7
PMID
22125095
发布时间
2024-02-04
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Neurotherapeutics
210-25页
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