Abacavir, didanosine and tenofovir do not induce inflammatory, apoptotic or oxidative stress genes in coronary endothelial cells.
第一作者:
Chul,Kim
第一单位:
Department of Cellular & Integrative Physiology, Indiana Center for Vascular Biology & Medicine, Indiana University School of Medicine, Indianapolis, IN, USA.
作者:
医学主题词
腺嘌呤(Adenine);抗HIV药(Anti-HIV Agents);细胞凋亡(Apoptosis);心血管疾病(Cardiovascular Diseases);细胞黏附分子(Cell Adhesion Molecules);冠状血管(Coronary Vessels);细胞因子类(Cytokines);去羟肌苷(Didanosine);双脱氧核苷类(Dideoxynucleosides);内皮细胞(Endothelial Cells);基因表达(Gene Expression);基因表达谱(Gene Expression Profiling);HIV感染(HIV Infections);HIV-1(HIV-1);人类(Humans);炎症(Inflammation);氧化性应激(Oxidative Stress);原癌基因蛋白质类(Proto-Oncogene Proteins)
DOI
10.3851/IMP1891
PMID
22155915
发布时间
2017-11-16
- 浏览6
Antiviral therapy
1335-9页
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