ETS1 transcriptional activity is increased in advanced prostate cancer and promotes the castrate-resistant phenotype.
第一作者:
A M,Smith
第一单位:
Department of Pathology and Laboratory Medicine, Medical University of South Carolina, Charleston, SC 29425, USA.
作者:
医学主题词
雄激素类(Androgens);细胞系, 肿瘤(Cell Line, Tumor);无病生存(Disease-Free Survival);人类(Humans);男(雄)性(Male);癌基因蛋白质v-akt(Oncogene Protein v-akt);睾丸切除术(Orchiectomy);表型(Phenotype);前列腺(Prostate);前列腺肿瘤(Prostatic Neoplasms);原癌基因蛋白质c-ets-1(Proto-Oncogene Protein c-ets-1);受体, 雄激素(Receptors, Androgen);信号传导(Signal Transduction);转录, 遗传(Transcription, Genetic);转录激活(Transcriptional Activation)
DOI
10.1093/carcin/bgs007
PMID
22232738
发布时间
2012-03-02
- 浏览27
Carcinogenesis
572-80页
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