Intracerebral lipopolysaccharide induces neuroinflammatory change and augmented brain injury in growth-restricted neonatal rats.
作者:
主题词
动物(Animals);动物, 新生(Animals, Newborn);细胞凋亡(Apoptosis);趋化因子CCL2(Chemokine CCL2);趋化因子CXCL1(Chemokine CXCL1);疾病模型, 动物(Disease Models, Animal);脑软化(Encephalomalacia);女(雌)性(Female);胎儿生长迟缓(Fetal Growth Retardation);人类(Humans);婴儿, 新生(Infant, Newborn);注射, 脑室内(Injections, Intraventricular);白质软化病, 脑室周围(Leukomalacia, Periventricular);脂多糖类(Lipopolysaccharides);胎盘功能不全(Placental Insufficiency);妊娠(Pregnancy);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley)
DOI
10.1038/pr.2012.26
PMID
22337231
发布时间
2022-04-18
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Pediatric research
645-52页
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