PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.
第一作者:
Dai-Fei,Wu
第一单位:
Ernest Gallo Clinic and Research Center, Department of Neurology, UCSF, Emeryville, California 94608, USA.
作者:
主题词
动作电位(Action Potentials);氨基酸取代(Amino Acid Substitution);动物(Animals);细胞, 培养的(Cells, Cultured);神经节, 脊(Ganglia, Spinal);痛觉过敏(Hyperalgesia);离子通道闸门(Ion Channel Gating);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);突变, 误义(Mutation, Missense);点突变(Point Mutation);蛋白激酶Cε(Protein Kinase C-epsilon);蛋白质加工, 转译后(Protein Processing, Post-Translational);大鼠(Rats);感觉感受器细胞(Sensory Receptor Cells);钠(Sodium);钠通道(Sodium Channels);应力, 物理(Stress, Mechanical);底物特异性(Substrate Specificity)
DOI
10.1172/JCI61934
PMID
22426212
发布时间
2021-10-21
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