Reduction of Na/K-ATPase potentiates marinobufagenin-induced cardiac dysfunction and myocyte apoptosis.
第一作者:
Changxuan,Liu
第一单位:
Department of Physiology and Pharmacology, University of Toledo, Toledo, Ohio 43614, USA.
作者:
医学主题词
动物(Animals);抗菌药(Anti-Bacterial Agents);细胞凋亡(Apoptosis);蟾酥甾类(Bufanolides);半胱氨酸天冬氨酸蛋白酶8(Caspase 8);半胱氨酸天冬氨酸蛋白酶9(Caspase 9);细胞, 培养的(Cells, Cultured);酶激活(Enzyme Activation);酶抑制剂(Enzyme Inhibitors);心脏病(Heart Diseases);小鼠(Mice);小鼠, 突变型(Mice, Mutant Strains);肌蛋白质类(Muscle Proteins);肌细胞, 心脏(Myocytes, Cardiac);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction);西罗莫司(Sirolimus);钠钾交换ATP酶(Sodium-Potassium-Exchanging ATPase);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);src族激酶类(src-Family Kinases)
DOI
10.1074/jbc.M111.304451
PMID
22451662
发布时间
2021-12-03
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